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In high-fat diet fed mice, treatment with the triagonist dose-dependently improved steatohepatitis and reduced levels of ALT, underpinning the potential for this compound to treat liver disease ( 3 ) agonist has shown to reverse metabolic syndrome related abnormalities ( 3 lowered ALT levels and improved macroscopic and histological features of NASH, including reversal of fibrosis ( Conclusion and future perspectives New knowledge about the pathophysiology of NAFLD has been accumulating over the last decade, displaying the complexity of the mechanisms involved in the development of this condition
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